Preliminary research show conolidine could inhibit particular ion channels, decreasing neuronal excitability and restricting ache signals. This mechanism is especially applicable in neuropathic pain, the place abnormal signaling brings about persistent soreness. Also, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to agony perception. Computational do... https://josephj256dmi8.wikipresses.com/user