Preliminary reports show conolidine could inhibit specific ion channels, reducing neuronal excitability and restricting agony signals. This mechanism is particularly relevant in neuropathic pain, exactly where irregular signaling triggers persistent pain. Also, conolidine seems to influence G protein-coupled receptor (GPCR) pathways integral to ache perception. Computational docking r... https://emperorx849lcs3.wikievia.com/user